young people suffering with heart problems?

Question

Hello everyone.

I am a 37 year old who has a heart condition (found out a couple of years ago). I also lost a brother from the same condition. Is there anyone else out there who would like to become a member of this group.

It's about Young Sudden Cardiac Deaths, and all other related topics, which will be starting soon. Any contributions would be greatly appreciated, as the aim is to make people aware of hidden
conditions in the under 35s. It's also about Support and friendship and especially for those of us who don't know about these hidden heart problems. PLEASE GEM UP!

Please come and say hello

all the best people

Los

http://uk.groups.yahoo.com/group/young_hearts_support/

Answer 1

I would be pleased to join ur group although I've no cardiac conditions but maybe I can help in support as u just said.
I'm a med student so maybe one day I'll be helpful for anny one in the group

Answer 2

HI i know this isnt for me but we recently found out my sister has a heart defect she has just turned 18.
She kept havin these seizure like attacks and she was sweaty and her heart beats so irregular at this time.
after being referred to the hospital by her doctor they ran loadsa tests the usuals:- ecg, eeg, 24hour tape, tilt test, BP etc...
They have now told her she has a leaky valve and that her heart misses a beat every hour and sometimes misses four beats, she will have to have a pace maker fitted and maybe corrective surgery, so we are all a bit bewildered

Answer 3

Here is an research article for everyone, hope every one on the forum will benefit from it, however I will join the group but the articles will be from professionals of high eminence, practicing in different part of the world.

Genetic risk factors in myocardial infarction at young age.

* Incalcaterra E,
* Hoffmann E,
* Averna MR,
* Caimi G.

Cardiology Section, University of Palermo, Palermo, Italy.

The role of genetic susceptibility to coronary artery disease (CAD) seems to be quite important in young patients. In the last years the attention has been focused on polymorphisms influencing some biological functions (coagulation and fibrinolysis, platelets, vascular function, lipid metabolism, inflammation). The study of prothrombotic polymorphisms has kindled a deep interest. The role of atherosclerosis and thrombosis is different in the different ages. In all the studies we examined, the polymorphism G20210A in the prothrombin gene was associated with an increased risk of acute myocardial infarction (AMI) in young people, especially when other risk factors were present. Contradictory results have been found in the studies on Factor V Leiden: according to many authors the activated protein C resistance (APCR) is associated with an increased risk of AMI only in smokers, above all if women. On the other hand, some polymorphisms of the Factor VII gene seem to be protective. Young AMI could be also caused by a reduction of the fibrinolytic activity, as it was found when the allele 4G in the promoter of plasminogen activator inhibitor (PAI) gene is present. The attention has also been focused on the effects of variations in genes that influence platelet functions. According to a metanalysis of studies published up to 1999, there is no association between the polymorphism PlA1/A2 of the GP IIIa gene and young AMI, whereas there is doubt about the role of the polymorphism in the GP IIb e GP Ib genes. Moreover, it seems to be present an association with the polymorphisms in the thrombopoietin gene (C4830A and A5713G). Also the role of some genes coding for proteins influencing the vascular functions has been valued. Few studies were performed on genetics of the renin-angiotensin-aldosterone system and the results are insufficient and contradictory, such as those about the association between the polymorphism G894T in the eNOS gene or the polymorphism C677T in the MTHFR gene and young AMI. Genes coding for proteins involved in the lipid metabolism have been closely examined. Many polymorphisms were discovered in the Apo B gene: the variant C-516T was found to be associated with increased LDL levels, whereas the results about the association between this and other polymorphisms in the same gene (I/D of LAL sequence, PvuII, MspI, Asp4311Ser) and young AMI are discordant. On the other hand, the variant e4 of the ApoE gene was associated with an increased risk of AMI at young age in many works. In the last years, a particular interest has kindled the study of the relationship between inflammation, atherosclerosis and CAD. Even if the studies performed are few, it was found an association between young AMI and polymorphism C-260T in the CD14 gene, between coronarics atherosclerosis and polymorphism A516C in the E Selectin gene or polymorphisms Leu125Val and Ser563Asn in the PECAM1 gene.

Answer 4

Hi! you sent me a link to that group. will join it when i get the chance to. I have a heart problem but it is not that bad. My brother also have a heart problem but a different condition.

Answer 5

Please see the web pages for more details on Sudden cardiac deaths in young athlets, Hypertrophic cardiomyopathy and Sudden adult death syndrome (Cardiac arrythmia)