Any MD or someone who knows precisely please explain to me exactly how edema of CHF develops.?

Question

I am a victim of severe edema of CHF post MASSIVE MI. Diuretics have NOT reduced my liver distended with water, nor my ankles to thighs bursting with water pressure in my tissues. (I have severely reduced left ventricular systolic function, & global hypokinesis) per radiologists , my Ejection fraction is 50%. I won't enter hospital since I am alone,uninsured, & they have no definite sure result to offer me but daily IV diuretics. Zaroxylyn has only a slight diuretic effect on me - but the water trapped in my ankles to thighs & liver is unaffected by drugs. So TELL ME - how does the severe EDEMA develop to begin with? & HOW can the diuretics draw out the water trapped in those areas? Or is this water affected at all by diuretics?
I begin to feel that the edema buildup in tissues is unaffected by drugs. AFter months of toxic drugs, I am not one bit less swollen with water & in pain from inner pressure. Can't flex my legs to wear a pants, climb a step. I'm unable to walk. Explain

Answer 1

considering ejection fraction of 50% this kind of edema is unnormal, using Digoxin might help your heart in pumping better, restrict your intake of water and salt, and taking enough diuretics such as Lasix(Furosemide) and hydrochlorotiazide help in minimizing the edem
Using special Varis Socks may help you.

Answer 2

do no longer DRINK greater WATER! ingesting greater water ought to easily reason pulmonary edema in you. you have a minimum of, left heart falure, probable from longterm high blood pressure. Ace bandages or TED hose will help, raise your ft and that i think of you're able to sense greater valuable. You do have lymph develop and probably seeing a D.O. or standard practitioner of osteopathy can help with lymph drainage. D.O's are no longer homeopaths and are liscenced in all 50 states and contributors of the AMA. maximum folk bypass to a minimum of one and don't even are conscious of it. maximum in overdue tiers of CHF get this and that i'm sorry and stable success.

Answer 3

my mom has edema and she used to seep alot. Have to wear kotex pads on her legs and all. Anyways they switched her heart medicine and it just drained off. I wish I knew the drug but maybe a different med will make the problem better. My mother has congestive heart failure and I presume you may too with your symptoms.

Answer 4

when you have cardiac problems the heart cannot push blood effectively, more and more blood accumulate in 1- ventricle so it become enlarged and stretch and then less effective to push blood, and 2- in veins of your body, when blood accumulate hydrostatic pressure in your veins increase so water is shift from veins to intracellular tissue which cause your problem, when you take diuretics it help ( in different ways it depend on type) in excretion water from your body, so it decrease pressure in vein and then decrease shifting of fluids, so you become better. take care.

Answer 5

Varicose vein can be a part of the game too. This article might help you:
Heart failure is the result of poor cardiac function and is reflected by a decreased volume of blood pumped out by the heart, the so-called cardiac output. Heart failure can be caused by weakness of the heart muscle, which pumps blood out through the arteries to the entire body, or by dysfunction of the heart valves, which regulate the flow of blood between the chambers of the heart. The diminished volume of blood pumped out by the heart (decreased cardiac output) is responsible for a decreased flow of blood to the kidneys. As a result, the kidneys sense that there is a reduction of the blood volume in the body. To counter the seeming loss of fluid, the kidneys retain salt and water. In this instance, the kidneys are fooled into thinking that the body needs to retain more fluid volume when, in fact, the body already is holding too much fluid.

This fluid increase ultimately results in the buildup of fluid within the lungs, which causes shortness of breath. Because of the decreased volume of blood pumped out by the heart (decreased cardiac output), the so-called effective blood volume in the arteries is decreased, despite the actual increase in the body's total fluid volume. An associated increase in the amount of fluid in the blood vessels of the lungs causes shortness of breath because the excess fluid from the lungs' blood vessels leaks into the airspaces (alveoli) in the lungs. This accumulation of fluid in the alveoli is called pulmonary edema. At the same time, accumulation of fluid in the legs causes pitting edema. This edema occurs because the build-up of blood in the veins of the legs causes leakage of fluid from the legs' capillaries (tiny blood vessels) into the interstitial spaces.

An understanding of how the heart and lungs interact will help you to better comprehend how fluid retention works in heart failure. The heart has four chambers; an auricle and a ventricle on the left side of the heart and an auricle and ventricle on the right. The left auricle receives oxygenated blood from the lungs and transfers it to the left ventricle, which then pumps it through the arteries to the entire body. The blood then is transported back to the heart by veins into the right auricle and transferred to the right ventricle, which then pumps it to the lungs for reoxygenation.

Left-sided heart failure, which is due primarily to a weak left ventricle, usually is caused by coronary artery disease, hypertension, or disease of the heart valves. Typically, when these patients initially come to the doctor, they are troubled by shortness of breath with exertion and when lying down at night (orthopnea). These symptoms are due to pulmonary edema that is caused by pooling of the blood in the vessels of the lungs. In contrast, right-sided heart failure, which often is due to chronic lung diseases such as emphysema, initially causes salt retention and edema. Persistent salt retention in these patients, however, may lead to an expanded blood volume in the blood vessels, thereby causing fluid accumulation in the lungs (pulmonary congestion) and shortness of breath. In patients with heart failure due to weak heart muscle (cardiomyopathy), both the right and left ventricles of the heart are usually affected. These patients, therefore, can initially suffer from swelling both in the lungs (pulmonary edema) and in the legs and feet (peripheral edema). The physician examining a patient who has congestive heart failure with fluid retention looks for certain signs. These include pitting edema of the legs and feet, rales in the lungs (moist crackle sounds from the excess fluid that can be heard with a stethoscope), a gallop rhythm (three heart sounds instead of the normal two due to muscle weakness), and distended neck veins. The distended neck veins reflect the accumulation of blood in the veins that are returning blood to the heart.