is atherosclerotic vascular disease caused by hypertension?

Question

my father recently passed of a heart attack. I just got the death certificate and it said hypertension which i know is high blood pressure and "atherosclerotic vascular disease" which I believe is hardening of the heart arteries. I assume the two are related diseases, in that one caused the other? anyone have any info? and can AVD be caused by inhaling harmful chemicals? like fumes on a job site or something?

Answer 1

Atherosclerosis and hypertension are directly related as are kidney disease and retinal disease . In general diet and hypertension team up to cause the hardening ,the HBP by injuring the inside of the ateries and the fat compounds by depositing at the site of the injuries in thick cheesy linings w.hich calcify and restrict blood flow to the heart .
Enviromental air pollution could cause lung ,liver kidney ,dermatological problems but not atherosclerosis

Answer 2

Hypertension can certainly contribute to atherosclerosis (which is often referred to as hardening of the arteries, it involves a buildup of cholesterol in the coronary arteries causing them to narrow), but there are many other factors as well such as cholesterol level, smoking history, genetics, and simple age. I don't know of any relation between enviornmental exposures (other than smoking) and atherosclerotic heart disease.
Now, that having been said, depending on who actually filled out the death certificate, it's probably best just to ignore it, those things are worthless. They are often filled out by a doctor who doesn't know the patient well who is making his best guess as to what may have caused the death, but doesn't know. Also, there are often other pressures, the cause of death must be accepted by the medical examiner, which is sometimes difficult (there are centain things that cannot be written, even if they're true) so often atherosclerotic heart disease is written when the doctor can't figure anything else, regardless of whether or not it reflects reality.

Answer 3

Atherosclerotic vascular disease refers to the narrowing and hardening of the arteries in the body. This process occurs naturally, to some degree, through aging.

Atherosclerosis leads to these disease complications:

1. Heart attack (coronary artery disease)
2. Stroke (vessel narrowing in the carotid arteries)
3. Peripheral vascular disease (narrowing of blood vessels to the lower extremities)
4. High blood pressure (promotes generalized atherosclerosis)
5. Kidney disease (vessel disease within the kidneys)
6. Abdominal Aortic Aneurysm (due to high blood pressure)
7. Ischemic Bowel disease

Risk factors are:

1. Diabetes
2. Smoking
3. Hypertension
4. High blood cholesterol

Answer 4

Primarily, it's the other way around. Hypertension contributes to kidney and vascular diseases! The kidneys autoregulate blood flow by controlling the diameter of tiny blood vessels going into and out of each of the microscopic urine-making structures called the "nephrons". When the pressure is high, these blood vessels become stiff from overgrowth of the muscle walls used to resist the flow. The nephrons themselves are damaged from pressure, and the organ's microstructure is slowly replaced by scar. High blood pressure also represents a challenge to the rest of the bodily blood vessels, which become thickened in response to the pressure, and which are prone to shearing injuries to the lining. This is especially true when the lining is infiltrated with cholesterol in the form of atherosclerotic plaques. As the blood vessels become thickened and stiff, the pulsations of flow hammer away at the walls, creating things like aneurysms (balloon-like dilations prone to rupture) and blockages where the atherosclerotic plaques rupture and clot. This latter mechanism is commonly the cause of heart attack and stroke, as well as other end-organ damage. There is one particular phenomenon, however, that DOES relate to your exact question. There is a condition known as renovascular hypertension, and this is one of the forms of high blood pressure that can be traced back to a treatable cause, and can be "cured" by an intervention. Most often, of course, hypertension is considered to be "essential hypertension" which means that there is no specific cause identified, and the hypertension itself needs to be treated because of the risks it poses to the patient's health. Renovascular hypertension is a problem that occurs in those people who develop flow limiting atherosclerotic plaques in the arteries leading off to the kidneys. When this happens, the flow to the kidneys is substantially decreased, and the kidneys are "fooled" into thinking that there is a problem of flow throughout the body. The kidneys react to a situation that, if the arteries to the kidneys were normal, would probably represent low circulating blood volume. Basically, the kidneys are unable to tell the difference between low flow because the arteries are bad, and low flow because the patient lost a lot of blood from an injury. The response from the kidney suffering low flow comes in the form of a hormone. This hormone is called "renin" and it has one job. Renin circulates in the blood and it turns a protein called "angiotensinogen" into "angiotensin 1". Angiotensin 1 is a hormone that increases the tension in blood vessels, but more importantly, it is a precursor to "angiontensin 2", which is what it its turned into immediately after passing through the lungs. Angiotensin 2 is one of the most powerful vaso-constricting hormones we have. It increases the blood pressure dramatically. Renovascular hypertension can be treated by procedures which open up the narrowed blood vessel, but the key is that the problem needs to be recognized first! Sometimes people with this problem go unrecognized for long periods of time. I hope that helps!

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Answer 5

I feel sorry for u're loss.....
hypertension is a risk factor for the formation of the atherosclerotic plaque...cuz high blood pressure damages the cellular lining of the vesseles (endothelium) on which the "fatty streaks" develope..which would progress into the atherosclerotic plaque...