What is vegetation of the cardiac valve ?

Answer 1

It means that there is a build-up of bacteria on your heart valve. Usually accompanied by high fevers of otherwise unknown source and caused by bacteriemia. Can be seen by echocardiogram.

Answer 2

Mitral Valve Vegetation

Answer 3

1

Answer 4

Aortic Valve Vegetation

Answer 5

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While that last answer is certainly correct, it may not be the easiest thing in the world to understand if you're not a medical professional, so let me summarize what the problems you're facing are. What it sounds like you're describing is an aortic valve vegetation with destruction of the valve leaflets. The heart valves are thin, flexible flaps of tissue that allow flow of blood in only one direction. When a leaflet gets infected, it can lead to local destruction of the valve itself leading to backflow of blood past the valve. Another big risk factor if you have an infectious vegetation on the aortic valve is for part of that infected vegetation to break off and go elsewhere in your bloodstream. That can lead to strokes (blockage of blood flow to a portion of the brain), loss of blood flow to the intestine or other end organ, or loss of blood flow to the extremities. All of these are medical or surgical emergencies. The backflow of blood can result in congestive heart failure due to fluid overload on the left ventricle with irreversible damage to the muscle. If the regurgitation is significant (in most cases of infectious endocarditis, it is) you will need a valve replacement surgery before congestive heart failure sets in. If you are already in congestive heart failure with irreversible heart damage, you will need a heart transplant. This is a decision that will need to be made by a cardiothoracic surgeon. Most inoperable patients will not live more than 6 months to a year. Symptoms can be controlled with diuretics and vasodilators to reduce cardiac work.

Answer 6

It is a bad bad thing to have. This is why certain heart pts have to take antibiotics before they have their teeth worked on. It is usually bacteria that has traveled from another source in the body to the heart valve. It sets up house there and destroys the heart valve. These pts go in antibiotics iv for a long time and then have their faulty heart valve replaced by a thoracic-cardiovascular surgeion.

Answer 7

Vegetation refers to an abnormal growth of tissue around a valve, composed of fibrin, platelets and bacteria. This can be a very serious condition and should not be taken lightly. Your physician can guide you towards the appropriate treatment.

Answer 8

The aim of this study was to determine the prevalence of cardiac valve disease in systemic lupus erythematosus or in patients with primary antiphospholipid syndrome and to assess the role of the antiphospholipid antibodies as risk factor for endocardial lesions. We studied 39 consecutive patients with systemic lupus erythematosus (mean age 34 +/- 12 years, 38 female and one male), 20 women with primary antiphospholipid syndrome (mean age 32 +/- 4 years) and 20 normal subjects (mean age 35 +/- 8 years, 15 female and five male). All patients with primary antiphospholipid syndrome had increased levels of serum anticardiolipin antibodies and recurrent fetal abortions; some of them also had arterial and/or venous thrombosis and/or thrombocytopenia. M-mode, two-dimensional and Doppler echocardiography were performed in all patients. IgG anticardiolipin antibodies were measured by an enzyme-linked immunosorbent assay. Valvular lesions were observed in 15 patients (38%) with systemic lupus erythematosus. These abnormalities included: mitral valve thickening or vegetation, mitral valve prolapse and aortic valve vegetation; mitral, aortic and tricuspid regurgitation; mitral stenosis. None of the patients with primary antiphospholipid syndrome and of the normal subjects was found to have valvular abnormalities. In systemic lupus erythematosus, high levels of anticardiolipin antibodies were detected in 73% of the patients with valvular lesions and in 67% of the patients without valvular lesions (P > 0.05). We conclude that valvular involvement is frequent in patients with systemic lupus erythematosus but it is apparently unrelated to antiphospholipid autoimmunization.