How do you know if you have "pattern baldness"?

Question

Hey 11 min and nobody knowes

HEY!!! this is a joke , answer it like a joke

finely ,but not there yet

Answer 1

The person standing next to you, is shaving the patterns...

Thank you for the web site "Junkyarddog.com"

Answer 2

Check to see if both your biological Grandfathers (Mother's and Father's Fathers) are bald. If neither is bald, you do not have "male pattern baldness." If one Grandfather is bald and the other is not, odds are only 50-50 you have "male pattern baldness." If both Grandfathers are bald, you have "male pattern baldness." However, having "male pattern baldness" does not automatically mean complete baldness. It has been proven that heavy doses of certain vitamins (such as B150 complex and plant sterols and stanols) taken over long periods of time can stop and in some cases reverse the effects of "male pattern baldness." And FYI, if you are a woman, same holds true, except that reference for baldness in women would be Grandmothers.

Answer 3

If after losing hair it never grows no matter what you do then I think that is pattern baldness. I have just guessed the answer. Maybe a doctor can help you better.

Answer 4

If the hair on your head spells the alphabet out in Upper and Lower case letters and then adds the numbers steadily one nunmber at a time.

Answer 5

When others are cutting their hair to look like yours.
Get it. Your head is the pattern for others to make copies of.

Answer 6

Although the classification for diagnosing pattern baldness is often a matter of subjectivity, one that has been in use since the 1970's is the Norwood's classification system. It has seven categories along with various subtypes for male pattern baldness, ordered in the extent of hair loss. Type seven, which describes the most severe and extensive of hair loss, is represented by "a narrow band of hair in a horseshoe shape survives on the side of the scalp."[1] This hair is soft and fine, and the hairs are also sparse on the nape of the neck and over both ears. The focus of this research paper will be on this type of pattern baldness; however, it is also important to note that a five-category classification for female pattern baldness, developed by Ebling and Rook, also exists. It takes into account that women could have both a diffuse form of hair loss in addition to a male pattern with recession of a frontetemporal hairline.[2]



Among Caucasians, 50 to 70% of the men and 40 to 50% of the women are affected by pattern baldness to some degree. For men, a rough and ready rule is that in their thirties, 30% have male pattern baldness. For men in their forties, 40% hair loss and so on until 70% of men are affected when 70 or more years old.2 Unlike men, the frequency of women affected does not continue to increase with increasing age.[3]



The American Indian and African American male populations have a lower incidence of pattern baldness compared to Caucasians. These frequencies suggest that genetic predisposition is an important factor in baldness susceptibility.[4] Total baldness is also much less common in the Chinese male population, but studies that compared Chinese and Chinese American subjects revealed a significant difference in expression frequency between the two populations. These studies suggest that environmental/dietary, not genetic, factors influence testosterone production and explain the difference between Caucasian and Chinese men. [5]





CAUSE AND ONSET



A number of environmental factors can influence pattern baldness: stress, chemotherapy, drugs, scalp infections, pulling hair. But by and large, the most common cause is still genetically programmed pattern baldness.[6] What are the direct expressions of these genes? Among the players involved in pattern baldness, or androgenetic alopecia (AGA), the one best understood belongs to a class of protein called 5-alpha reductase. Normally found in the prostate, this protein reduces testosterone to dihydrotestosterone (DHT). During development, DHT initiates the differentiation of of prostate and male external genital. In contrast, 5-alpha reductases found in the scalp and elsewhere on the skin, where the enzyme catalyzes the same reaction as in the prostate, are associated with acne, hirsutism (hairy-ness), and, of course, alopecia (baldness).[7] Age is the determining factor that triggers the cells in the dermal papillae, which are fingerlike structures that project into the epidermis, to begin synthesizing 5-alpha-reductase. In addition, the level of testosterone rises in male after puberty, as one may recall from the good O' hormone-raging days of high school. As a result, the dermal papillae, being highly vascular, or having many capillaries looped through, are continuously bathed in these circulatory testosterones. In women, if excessive endocrine activity produces more testosterone than the body needs, the capillaries would also be rich with androgens. [8]



What role does 5-alpha reductase play in AGA? First, this enzyme converts the androgens into DHTs; DHTs are then absorbed into the cells of hair follicle, a highly vascular, bulb shaped depression in the scalp. Once inside the follicle cell, DHTs will bind to cytoplasmic receptors, forming the so-called DHT-receptor complexes. And as secondary messengers, these complexes effect the onset of hair miniaturization.[9]





MINIATURIZATION



Miniaturization is essentially the shrinkage in length and thickness of hair and hair follicles, as well as the shortening of anagen phase, the active hair growth phase of the hair growth cycle. Normally, cells on the bottom of the follicle, collectively called the mesodermal tissue, divide and form hair. New hair grows along the stem of the follicle, replacing shed hair. During miniaturization, however, DHT-receptor complexes slow down hair production in the mesodermal tissue and cause the tissue to produce weaker, shorter hair. Eventually, DHT-receptor complexes may even halt terminal hair growth altogether, leaving nothing behind except a microscopic forest of fuzzy vellus hair. Balding individuals do not get a replacement hair for the ones that are shed.





REFERENCE







--------------------------------------------------------------------------------

[0] http://healthfactsandfears.com/featured_articles/feb2002/baldness020102.html Accessed 01/15/03

[1] http://www.keratin.com/ac/baldnesspatterns/baldnessclassification/002norwoodbaldnessclassification.

shtml Accessed 01/15/03

[2] http://www.keratin.com/ac/baldnesspatterns/baldnessepidemiology/001malebaldnessiscommon.shtml Accessed 01/15/03

[3] http://www.keratin.com/ac/baldnesspatterns/baldnessepidemiology/002femalebaldnessiscommon.shtml Accessed 01/15/03

[4] http://www.keratin.com/ac/baldnesspatterns/baldnessepidemiology/003ethnicgroupsbaldnessfrequency.sh tml Accessed 01/15/03

[5] Santner SJ, Albertson B, Zhang GY, Zhang GH, Santulli M, Wang C, Demers LM, Shackleton C, Santen RJ. Comparative rates of androgen production and metabolism in Caucasian and Chinese subjects. J Clin Endocrinol Metab 1998 Jun;83(6):2104-9

[6] Caine, WK, Garfinkel P. The Male Body: An Owner's Manual. Pennsylvania: Rodale Press; 1996. 405 p.

[7] http://www.keratin.com/aa/aa009.shtml Accessed 01/15/03

[8] http://www.helioshealth.com/mpb/causes.html#Anchor-Hormone-220 Accessed 1/15/03.

[9] http://www.thymusking.com/hair_loss.htm Accessed 01/15/03

Answer 7

It depends on what kind of a patern it makes.

Answer 8

because it runs in your family

Answer 9

when your head is starting to look like a flannel shirt? lol idk