My face is sensitive and is red.What can i do I'v tried everything and it back fires.I NEED HELP REALLY BAD!!!

Question

face is red,flaky really sensitive sometime it hurts.acne is also involved but really lite not to much.

Answer 1

try and go to your dermatologist. it`ll help.

Answer 2

That flaky is your face is real dry, you need moisturizers on all the time. Wash you face in sensitive dove soap, then take a cold water wash cloth and apply it to your face, let it stay a minute, that closes the pores of your skin. I use Burts Bees products they are all natural. when you go to bed at night when you take your bath or shower wash your face with sensitive dove and wash it of with cold water. Put a burts bees night moisturizing on your face and neck. The next morning put daytime burts bees moisturizing creme and then apply burts marshmallow vanishing creme it has mango butter. Do this every night and every morning and during the day until it is under control if your face feels dry, repeat what you did in the morning. with both cremes. You will find your face will after a short period be real smooth and no redness. Keep doing this and you will have a beautiful smooth skin. Burts is made out of all natural get the moisturizing tinted facial creme for your makeup and burts blush.
Every store has a good line of this product on the Internet for the best prices on all the products go to Advantage Nutrition and you can get everything. Just hit Burts Bees.because they have other natural products, also

Answer 3

If you have health insurance, I suggest seeing a dermatologist...they are wonderful. Or maybe have you tried a mousturizer for sensitive skin? I've heard of something before that causes red blotchy skin, but I don't think theres anything over the counter for it. Good luck!!

Answer 4

I would seriously recommend a dermatologist! They could check your skin and then could tell you what is wrong, and exactly what you need to use or what to do. You don't at this point want to experiment with things or it might only get worse. If you cant get to a dermatologist, I would even go to a pharmacy and talk to a pharmacist, and sometimes they are helpful. Good luck!

Answer 5

Rosacea is a skin disease that causes redness, pimples, and red lines on the nose, cheeks, chin, and forehead. A rash over the cheeks, nose, forehead, and chin often occurs. People sometimes call it "adult acne" because it can cause pimple-like outbreaks. Rosacea can also cause burning and soreness in the eyes and eyelids.

You may control rosacea with medication and by avoiding triggers that lead to flare-ups. Left untreated, rosacea can get worse. Large, disfiguring bumps on the nose and face and serious eye problems are signs of severe rosacea.

Fair-skinned people between the ages of 30 and 60 are most likely to develop rosacea. Women are more likely to have rosacea, but men usually have more severe symptoms.

Answer 6

Use Neutrogena Deep Clean Invigorating Scrub; it will have your skin looking cleaner right after you wash your face. Then throughout the day, use Ponds Clean Sweep Makeup Removing Wipes, and then reapply your makeup. I know that's dumb since you go through more makeup, but your skin will be/should be cleaner.

Answer 7

I have no acne but my skin goes flaky so the best products I found were Rosemary Products for my face, I have real sensitive skin too.

Answer 8

Cetaphil is a brand of soaps and lotions that is excellent for irritated skin and can be found in drugstores.

Answer 9

it is prbably contact dermatites(dont worry its not as bad as it sounds.!) i say just moisturize it! if you can go to the dermatologist!


Description
Only the superficial regions of the skin are affected in contact dermatitiis. Inflammation of the affected tissue is present in the epidermis (the outermost layer of skin) and the outer dermis (the layer beneath the epidermis) (ESCD 2006)[1].

Unlike Contact Urticaria in which a rash appears within minutes of exposure and fades away within minutes to hours, contact dermatitis takes days to fade away and then only if the skin no longer comes in contact with the allergen or irritant.[2] Chronic contact dermatitis can develop where the removal of the offending agent no longer provides expected relief.

In North America, the most common cause of contact dermatitis is poison ivy, including poison oak and sumac. Other common causes are harsh soaps, detergents, and cleaning products.


Types of Contact Dermatitis
There are three types of contact dermatitis: irritant contact, allergic contact, and photocontact dermatitis. Photocontact dermatitis is divided into two catergories: phototoxic and photoallergic.

Irritant Contact Dermatitis (ICD)
This is the most common form of contact dermatitis, affecting around 1-2% of healthy Europeans (ESCD 2006), and can be caused by either an acute or chronic exposure to a toxic insult. It is also referred to as non-allergic contact dermatitis (along with traumiterative or housewives eczema) referring to its non-immunological mechanism of toxicity and can be caused wither by chemical or physicalirritants. The following definition is provided by Mathias and Maibach (1978):[3]

a nonimmunologic local inflammatory reaction characterized by erythema, edema, or corrosion following single or repeated application of a chemical substance to an identical cutaneous site
Chemical Irritant Contact Dermatitis (CICD) can be subdivided into acute and chronic ICD which are usually associated with strong and weak irritants respectively (HSE MS24)[4]. The mechanism of action varies between toxins. Detergents, surfactants, extremes of pH and organic solvents all have the common effect of directly affecting the barrier properties of the epidermis. These include removing fat emulsion, inflicting cellular damage on the epithelium, or increasing the transepidermal water loss by damaging the horny layer water-binding mechanisms and damaging the DNA which causes the layer to thin. As suggested previously, strong concentrations of irritants cause an acute effect, but this is not as common as the accumulative, chronic effect of weaker irritants whose deleterious effects build up with subsequent doses

Physical irritant contact dermatitis (PICD) is a less well researched form of ICD (Maurice-Jones et al)[5]. This is due to its highly varied and numerous mechanisms of action as well as a lack of a test for its diagnosis. A complete patient history combinated with negative allergic patch testing is usually necessary to reach a correct diagnosis. The simplest form of PICD results from prolonged rubbing, although the diversity of implicated irritants is far wider.[citation needed]

Many plants cause ICD by directly irritating the skin. Some plants act through their spines or irritant hairs. Some plant such as the buttercup, spurge and daisy, act by chemical means. The sap of these plants contains a number of alkaloids, glycosides, saponins, anthroquinones, and in the case of plant bulbs, irritant calcium oxalate crystals, all of which can cause CICD (Mantle and Lennard, 2001).


Allergic Contact Dermatitis (ACD)
This condition is the manifestation of an allergic response caused by contact with a substance. A list of common allergens is shown in Table 1 (Kucenic and Belsito, 2002)[7]. Although less common than ICD, it is accepted to be the most prevalent form of immunotoxicity found in humans (Kimbe et al 2002)[8]. By its allergic nature, this form is a hypersensitive reaction which is atypical within the population. The mechanisms by which these reactions occur are complex, with many levels of fine control. Their immunology centres around the interaction of immunoregulatory cytokines and discrete subpopulations of T Lymphocytes.

ACD arises as a result of two essential stages - an induction phase which primes and sensitises the immune system for an allergic response, and an elicitation phase in which this response is triggered (Kimbe et al 2002). As such, ACD is termed a Type IV delayed hypersensitivity reaction involving a cell-mediated allergic response. Contact allergens are essentially soluble haptens (low in molecular weight) and, as such, have the physico-chemical properties that allow them to cross the stratum corneum of the skin. They can only cause their response as part of a complete antigen, involving their association with epidermal proteins forming hapten-protein conjugates. This in turn requires them to be protein-reactive.

The conjugate formed is then recognised as a foreign body by the Langerhans Cells (LC) (and in some cases Dentritic Cells (DC)), which then internalise the protein, transport it via the lymphatic system to the regional lymph nodes and present the antigen to T-lymphocytes. As mentioned earlier, this process is controlled by cytokines and chemokines, with tumour necrosis factor alpha (TNF-α) and certain members of the interleukin family (1, 13 and 18) and their action serves either to promote or to inhibit the mobilisation and migration of these LCs. (Kimbe et al 2002) As the Langerhans cells are transported to the lymph nodes, they become differentiated and transform into Dentric Cells which are immunostimulatory in nature.

Once within the lymph glands, the differentiated DCs act to present the allergenic epitope associated with the allergen to T lymphocytes. As a result, these T cells divide and differentiate, clonally multiplying so that if the allergen is experienced again by the individual, these T cells will respond more quickly and more aggressively.

As mentioned briefly, the immunological reaction involved in ACD is highly complex with many levels of fine control. Kimbe et al (2002) explore these complexities in short. It appears that there are two major phenotypes of cytokine production (although there exists a gradient of subsets in between), and these are termed T-helper 1 and 2 (Th1 and Th2). Although these cells initially differentiate from a common stem cell, they develop with time as the immune system matures. Th1 phenotypes are characterised by their focus on Interleukin and Interferon, while Th2 cells action is centred more around the regulation of IgE by cytokines. The CD4 and CD8 T lymphocyte subsets also have been found to contribute to differential cytokine regulation, with CD4 having been shown to produce high levels of IL-4 and IL10 while solely CD8 cells are associated with low levels of IFN?. These two cell subtypes are also closely associated with the cell matrix interactions essential for the pathogenesis of ACD.

It has been suggested (White et al 1986) [9] that there appears to be a threshold to the mechanisms of allergic sensitisation by ACD-associated allergens. This is thought to be linked to the level at which the toxin induces the up-regulation of the required mandatory cytokines and chemokines. It has also been proposed that the vehicle in which the allergen reaches the skin could take some responsibility in the sensitisation of the epidermis by both assisting the percutaneous penetration and causing some form of trauma and mobilisation of cytokines itself.


Photocontact Dermatitis (PCD)
Sometimes termed "photoaggravated"(Bourke et al 2001)[10], and divided into two categories, phototoxic and photoallergic, PCD is the eczematous condition which is triggered by an interaction between an otherwise unharmful or less harmful substance on the skin and ultraviolet light (320-400nm UVA) (ESCD 2006), therefore manifesting itself only in regions where the sufferer has been exposed to such rays. Without the presence of these rays, the photosensitiser is not harmful. For this reason, this form of contact dermatitis is usually associated only with areas of skin which are left uncovered by clothing. The mechanism of action varies from toxin to toxin, but is usually due to the production of a photoproduct. Toxins which are associated with PCD include the psoralens. Psoralens are in fact used therapeutically for the treatment of psoriasis, eczema and vitiligo.

Photocontact dermatitis is another condition where the distinction between forms of contact dermatitis is not clear cut. Immunological mechanisms can also play a part, causing a response similar to ACD.


Prevention
Since contact dermatitis relies on an irritant or an allergen to initiate the reaction, it is important for the victim to identify the responsible agent and avoid it. This can be accomplished by having patch tests, a method commonly known as allergy testing. The victim must know where the irritant or allergen is found to be able to avoid it. It is important to also note that chemicals sometimes have several different names. This doesn't always have a great success rate. [11]


Summary
The distinction between the various types of contact dermatitis is based on a number of factors. The morphology of the tissues, the histology, and immunologic findings are all used in diagnosis of the form of the condition. However, as suggested previously, there is some confusion in the distinction of the different forms of contact dermatitis (Reitschel 1997)[12]. Using histology on its own is insufficient, as these findings have been acknowledged not to distinguish (Rietschel, 1997), and even positive patch testing does not rule out the existence of an irritant form of dermatitis as well as an immunological one. It is important to remember, therefore, that the distinction between the types of contact dermatitis is often blurred, with, for example, certain immunological mechanisms also being involved in a case of irritant contact dermatitis.


websites to go to for info and such:
-http://www.emedicinehealth.com/contact_dermatitis/article_em.htm

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http://www.nlm.nih.gov/medlineplus/ency/article/000869.htm

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http://allergies.about.com/cs/skin/a/aa030899.htm

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http://www.thedoctorsdoctor.com/diseases/contact_dermatitis.htm







i hope that i helped! again dont worry it will heal and won't leave a mark!! i had it before and it will prabably go away soon! my story is kinda funny actually! i was at camp for a week and it was called challenge camp for catholic girls well when i came back i noticed a rash on my face. the thing was , that it was in the shape of the cross!!!! it was so cool! it was very ironic!
also i got it when i put body wash on my face (dont ask it was a accident and it smelled really good! hahahaha!) but it will prabably go away b4 u go back to school from the holidays!

good luck!bye! i hope this helped!