What is thyroiditis and how is it treated?

Answer 1

This going to be a long post but will answer the bulk of your questions. Hope it helps. This is taken from a powerpoint lecture on Thyroiditis so the formatting may be off.

Thyroiditis

Inflammation of the Thyroid Gland


Thyroiditis is an inflammation (not an infection) of the thyroid gland. Several types of thyroiditis exist and the treatment is different for each.


Hashimoto's Thyroiditis. Hashimoto's Thyroiditis (also called autoimmune or chronic lymphocytic thyroiditis) is the most common type of thyroiditis. It is named after the Japanese physician, Hakaru Hashimoto, that first described it in 1912. The thyroid gland is always enlarged, although only one side may be enlarged enough to feel. During the course of this disease, the cells of the thyroid becomes inefficient in converting iodine into thyroid hormone and "compensates" by enlarging (for a review of this process see our function page). The radioactive iodine uptake may be paradoxically high while the patient is hypothyroid because the gland retains the ability to take-up or "trap" iodine even after it has lost its ability to produce thyroid hormone. As the disease progresses, the TSH increases since the pituitary is trying to induce the thyroid to make more hormone, the T4 falls since the thyroid can't make it, and the patient becomes hypothyroid. The sequence of events can occur over a relatively short span of a few weeks or may take several years.

Treatment is to start thyroid hormone replacement. This prevents or corrects the hypothyroidism and it also generally keeps the gland from getting larger.

In most cases the thyroid gland will decrease in size once thyroid hormone replacement is started.

Thyroid antibodies are present in 95% of patients with Hashimoto's Thyroiditis and serve as a useful "marker" in identifying the disease without thyroid biopsy or surgery.

Thyroid antibodies may remain for years after the disease has been adequately treated and the patient is on thyroid hormone replacement.


De Quervain's Thyroiditis. De Quervain's Thyroiditis (also called subacute or granulomatous thyroiditis) was first described in 1904 and is much less common than Hashimoto's Thyroiditis. The thyroid gland generally swells rapidly and is very painful and tender. The gland discharges thyroid hormone into the blood and the patients become hyperthyroid; however the gland quits taking up iodine (radioactive iodine uptake is very low) and the hyperthyroidism generally resolves over the next several weeks.

Patients frequently become ill with fever and prefer to be in bed.

Thyroid antibodies are not present in the blood, but the sedimentation rate, which measures inflammation, is very high.

Although this type of thyroiditis resembles an infection within the thyroid gland, no infectious agent has ever been identified and antibiotics are of no use.

Treatment is usually bed rest and aspirin to reduce inflammation.

Occasionally cortisone (steroids) (to reduce inflammation) and thyroid hormone (to "rest" the thyroid gland) may be used in prolonged cases.

Nearly all patients recover and the thyroid gland returns to normal after several weeks or months.

A few patients will become hypothyroid once the inflammation settles down and therefore will need to stay on thyroid hormone replacement indefinitely.

Recurrences are uncommon.


Silent Thyroiditis. Silent Thyroiditis is the third and least common type of thyroiditis. It was not recognized until the 1970's although it probably existed and was treated as Graves' Disease before that. This type of thyroiditis resembles in part Hashimoto's Thyroiditis and in part De Quervain's Thyroiditis. The blood thyroid test are high and the radioactive iodine uptake is low (like De Quervain's Thyroiditis), but there is no pain and needle biopsy resembles Hashimoto's Thyroiditis. The majority of patients have been young women following pregnancy. The disease usually needs no treatment and 80% of patients show complete recovery and return of the thyroid gland to normal after three months. Symptoms are similar to Graves' Disease except milder. The thyroid gland is only slightly enlarged and exophthalmos (development of "bug eyes") does not occur. Treatment is usually bed rest with beta blockers to control palpitations (drugs to prevent rapid heart rates). Radioactive iodine, surgery, or antithyroid medication is never needed. A few patients have become permanently hypothyroid and needed to be placed on thyroid hormone.

Answer 2

The thyroid is a butterfly-shaped gland that sits low on the front of the neck. Your thyroid lies below your Adam’s apple, along the front of the windpipe. The thyroid has two side lobes, connected by a bridge (isthmus) in the middle. When the thyroid is its normal size, you’re unlikely to be able to feel it.
All types of hyperthyroidism are due to overproduction of thyroid hormones, but the condition can develop in several ways.
Thyroid hormone production can be suppressed or halted completely in these ways: https://tr.im/gLIxf

Answer 3

I suffered 5 years for hypothyroid and I definetly cure my disease with this natural treatment http://treatment-hypothyroidism.keysolve.net

Two things you want to consider as problems: Toxicity and Autoimmunity (or both)

#1 Toxicity. You mentioned black lines in your gums. This is a classic presentation for lead toxicity. This would also explain problems with finger nails (though a thyroid problem would do the same. Many think exposure is limited to paint used before 1978, but lead is still being used in many different things (just look at all the toy recall...very sad).
This can be diagnosed by blood, hair and/or stool analysis.

#2. Autoimmune means that your body's immune system is attacking itself, in your case the thyroid gland.
It does this by recognizing certain protein strands and tagging it with antibodies. Then your body sends out it seek and destroy immune mediators to kill off the tagged protein structures. Usually this is how your body defends itself from viruses, bacteria, fungi, ect. There is a problem with your tagging system that has caused your body to attack itself. This is usually diagnosed by testing for antibodies in blood work, as well as clinical symptoms.

How does this happen? There is no one pin pointed cause. The most popular reason is because of some break down in your filtering system. You normally keep bad things out through things like your skin, gut lining etc. If there is a problem with your gut, things get through that shouldn't, your body recognizes it as foreign, tags it and destroys it. Sometimes the protein structures of the things that get through look like the protein structures that make up certain cells in your body. That is why you see some of the other post saying that this autoimmune disorder is related to other autoimmune disorders (diabetes, lupus, RA, etc). It is really a problem with your filter, not your thyroid (or at least primarily the problem). Treating the thyroid might help somewhat, but it will not address the problem (in my example, the gut lining (filter) being disrupted).

Answer 4

Thyroiditis is an inflamation of the thyroid gland which is located in front of the neck. Your doctor will deal with this for you.

Answer 5

inflammation of the thyroid gland, usually cause by viral infection.
Heal by itself, but need medical attention, in case of the danger of a thyroid crisis.

Answer 6

Thyroditis is an autoimmune disease with enlargement of thyroid and infiltration by lymphocytes. Hypothyroidism occurs with it. There are more types, though, histologically.

Answer 7

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Answer 8

Thyroiditis refers to an inflammation of the thyroid gland.
click here:http://en.wikipedia.org/wiki/Thyroid_gland#Diseases_of_the_thyroid_gland

Answer 9

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Answer 10

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