Is there any medicinal cure for AIDS?

Question

Means Some cure or antibiotic

Answer 1

Do you read newspapers? or may listen or watch news on radio TV?

Answer 2

AIDS is caused by the HIV virus, and there is NO virus on Earth which we can cure - anything you see in the movies or on TV about viral cures, or 'anti-viruses' (such as in Mission Impossible 2) is complete and utter rubish!! Viral infections such as smallpox have been erradicated through the use of vaccines, preventing the spead of the disease, rather than curing those infected.

We have anti-viral drugs, but these do not cure the patient of the virus. They work by supressing the ability of the virus to replicate. With some infections, 'slowing down' the virus in this way can allow the body's own immune system to then cure itself of the virus. But ultimately, the drugs are simply a 'helping hand'.

However, the problem with HIV and AIDS is that the virus attacks the immune system itself. So, we can't cure it, and neither can out own bodies. We do now have effective anti-viral dugs for managing AIDS ('bkzalley' explains this in detail). The patient will have a greatly increased life-span, and a much better quality of life. However, they will always have the virus, and as such, can always pass it on!

However, these drugs are only available to a minority of people - Third World Countries cannot afford the high prices, and the drug companies refuse to sell them at a cheaper rate. As such, HIV continues to be a pandemic in these areas.

Answer 3

There is no cure for AIDS at this time. However, a variety of treatments are available that can delay the progression of disease for many years, and improve the quality of life of those who have developed symptoms.

Antiretroviral therapy suppresses the replication of the HIV virus in the body. A combination of several antiretroviral agents, termed highly active antiretroviral therapy (HAART), has been highly effective in reducing the number of HIV particles in the blood stream, as measured by a blood test called the viral load. This can help the immune system recover from the HIV infection and improve T-cell counts.

Although this is not a cure for HIV, and people on HAART with suppressed levels of HIV can still transmit the virus to others through sex or sharing of needles, these treatment have been enormously effective for the past ten years. There is good evidence that if the levels of HIV remain suppressed and the CD4 count remains high (above 200), life can be significantly prolonged and improved. However, HIV may become resistant to HAART in patients who do not take their medications on schedule every day. Genetic tests are now available to determine whether a particular strain is resistant to a particular drug -- these may be useful in determining the best drug combination, and adjusting the regimen if it starts to fail. These tests should be performed for any failing treatment course, and prior to starting therapy.

When HIV becomes resistant to HAART, salvage therapy is required, to try to suppress the resistant strain of HIV. Different combinations of medications are used to try to reduce viral load, and there are a variety of new drugs coming out on the market for the treatment of drug-resistant HIV.

Treatment with HAART is not without complications. HAART is a collection of different medications, each with its own side effects. Some common side effects are nausea, headache, weakness, malaise, and fat accumulation on the back and abdomen ("buffalo hump"). When used long-term, these medications increase the risk of heart attack by affecting fat breakdown, specifically through increasing lipids and glucose levels.

Any doctor prescribing HAART should carefully follow the patient for possible side effects associated with the combination of medications the patient takes. In addition, routine blood tests measuring CD4 counts and HIV viral load (a blood test that measures how much virus is in the blood) should be taken every three to four months. The goal is to get the CD4 count as close to normal as possible, and to suppress the HIV viral load to an undetectable level.

Other antiviral agents are in investigational stages and many new drugs are in development. In addition, growth factors that stimulate cell growth, such as Epogen (erthythropoetin) and G-CSF are sometimes used to treat anemia and low white blood cell counts associated with AIDS.

Medications are also used to prevent opportunistic infections (such as Pneumocystis carinii pneumonia) if the CD4 count is low enough . This keeps AIDS patients healthier for longer periods of time. Opportunistic infections are treated as they occur.

Answer 4

the full form of AIDS is Acquired Immune Deficiency Syndrome.
there is no cure for aids. preventive measures r the only cure.

Answer 5

Eat 4 to 5 leaves of tulsi with water every day. Tulsi will help ful in enhancing your imune system.
Watch Astha Channel every day 5 to 7 am ( IST) and at night 8 to 9 pm (IST) and do Pranayam daily.

Answer 6

this report will give u some idea for the cure of aids

THE Human Immunodeficiency Virus (HIV), the causative agent of Acquired Immune Deficiency Syndrome (AIDS), which has infected an estimated 30 million people around the world, is perhaps the cleverest of the micro-organisms that afflict humans. As the names of the virus and the disease imply, the infection attacks the very root of the immune system, namely the T-cell lymphocytes (white blood cells that normally confer protection). It does not so much destroy the immune system as it thrives in it. Ongoing research seems to reveal that HIV usurps the immune response for its own replication in the human host; this situation makes both prophylactic and therapeutic interventions major challenges to biomedical research.



COURTESY: USIS
A finding by Dr. Anthony S. Fauci (right) and his team of researchers in the U.S. may lead to major advances in the treatment of AIDS.

In a major research finding reported at the ICI, Fauci's team has identified one such hideout. The team has also found a therapeutic mechanism to flush out the virus pool from there and purge it with the help of anti-retroviral drugs. However, as Fauci cautioned, the discovery does not mean that a cure is at hand. "First we have looked at only one reservoir whereas there could be many. Furthermore, even here we have to see that the viral load does not increase in the long term when the patients are put off drug therapy. This requires a carefully planned follow-up strategy," he said.

The drug regimen that has been found to work reasonably well, and is followed all over the world, is the Highly Active Anti-Retroviral Treatment (HAART), a combination of zidovudine (AZT), lamivudine (3TC) and crixivan (Indinavir). It is basically a dual approach in the sense that it uses two types of drugs, one a reverse transcriptase (RT) inhibitor and the other a protease inhibitor, to prevent virus replication and/or viral protein expression. Reverse transcriptase is the enzyme that helps the RNA virus to convert into a DNA form and integrate itself into the host cell DNA so that it can replicate. But HIV has been found to mutate in order to evade any intervention at this stage. Protease is the enzyme which aids in the maturation of the virus before the viral proteins can be expressed. The assumption underlying the therapy is that it is extremely unlikely that a virus will mutate in the same cell and at the same time to evade both RT and protease inhibitors. Therefore, closing both the gates should work. The regimen includes two RT inhibitors and one protease inhibitor. But the therapy fails to attack the latent reservoirs.

One of the "reservoirs" of HIV is believed to be the cells known as 'Resting CD4+ T cells', which are part of the immune system. CD4+ T cells are those lymphocytes which express on their surface the protein called CD4 that binds well to HIV. Resting CD4+ T cells are those among the CD4+ T cells which have not yet seen the target - the antigenic molecules - and hence have not so far been activated to take part in the immune response. The body has a total of about a trillion lymphocytes of which 'Resting CD4+ T Cells' that harbour 'replication competent HIV' constitute tens to hundreds of millions. The important thing is that this latent pool is formed immediately after the first infection and can remain latent for as long as 15 years.

One of the clinical observations that led to Fauci's research is that in patients with active infection, any intervention to treat (or prevent) opportunistic infections such as tuberculosis was found to enhance the circulating viral load in the body. He inferred that the cytokines (or regulatory molecules) such as Interleukin-2 (IL-2) that control proliferation, differentiation and the immune response of a number of immune system cells, notably T cells, seem to drive the replication of HIV from the latently infected cells. Fauci's team found that cytokines not only regulated the replication of the latent virus pool but enhanced the expression of latently infected cells. In other words, cytokines such as IL-2 seem to clear the virus out of its hideout in resting CD4+ cells and the virus then begins to replicate and express itself. The exact mechanism by which this happens is not quite clear yet but this suggested that if HAART is adminstered to HIV/AIDS patients in combination with IL-2, the latter could tease the virus pool from its hiding and purge it with the action of HAART.

The NIAID team studied two groups of patients: 12 patients receiving only HAART and 14 patients receiving a combination of IL-2 and HAART. IL-2 was administered either subcutaneously or intravenously; the dose was between three million and 18 million international units (IUs) a day during a five-day treatment cycle, which was followed by a rest period of eight weeks before the next treatment cycle began. The viral load before the start of the experiment was less than 50 copies of HIV per millilitre of blood. The median time that the patients in the IL-2 + HAART group were receiving IL-2 was 39 months.

In six of the 14 patients (42.8 per cent), the researchers did not find any detectable replication-competent HIV in 10-20 million resting cells taken from the patients' peripheral blood. When much larger numbers of resting cells were cultured, in the range of between 200 and 300 million, three of the six did not show any detectable replication-competent virus. By contrast, in patients receiving HAART alone, replicating virus was found consistently in all the 12 patients. In the former group, a biopsy of the lymph-node tissue (the seat of the lymphocytes) also showed no detectable replication-competent HIV. When patients responded well, the interval between cycles of IL-2 was extended. The NIAID study now has patients who require only IL-2 once in several months and even some who require it only once or twice a year. One of the patients in the study has responded so well that he has received IL-2 only once in the past two years.

Answer 7

there is no cure, but a vaccine is being developed in the US

Answer 8

there is temprorar cure but not permanent cure

Answer 9

You're kiffing? no. there is not.

Answer 10

No.