sleep question.?

Question

have u heard of the 5 stages of sleep?
Well, my question is what happens if u wake up to pee, drink water,or just because?do the stages have to start all over again or do they continue.if i sleep for hours then get up then lay back down for another hours,do we go through all the stages still?
i know it's a weird question but ihave always been curious.

Answer 1

If your sleep is interrupted by something simple, like a bathroom break, and you don't have any sleep disorders, you'll quickly fall back through the stages to where your body is used to being at that time. If you've already slept the number of hours your body requires, and you force yourself to go back to sleep, your deep sleep cycles will be much lighter, or may not happen at all.

Answer 2

well as you say, there are different stages on sleeping, and these stages are measured by your brainwave activity!

If,for example, you are in the deep sleep stage, and u go and take a pee...your brain becomes active for a short period of time (in other words..while u are "Awake") after you pee and go back to sleep, your brain quickly relaxes and goes back to the deep sleep stage.

If however, u wake up and go watch tv for 30 minutes or so...your brain becomes highly active again, and then YES, you would have to go through all the stages of sleep again

Answer 3

wouldnt it depend on how deep your sleep falls in the hour? but who knows they probably havent done a study on that yet. I'd like to know the answer. I know if I do that, I can fall asleep easier, and I know I have dreampt after that. which dont they say that rem stage is dreaming?

Answer 4

just like u said it is a cycle. if u wake up,then go back to sleep it starts all over. check any sleep apnea site for more info

Answer 5

The cycle between sleep and wakefulness involves different stages of sleep. Currently, scientists divide sleep into two general types: REM (Rapid Eye Movement) and NREM (non-REM). REM sleep is characterized by desynchronization of the electroencephalograph (EEG), loss of skeletal muscle tone, and sympathetic nervous system; whereas non-rapid eye movement sleep is characterized by parasympathetic nervous system activity (Legramante & Galante, 2005).

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Stages of sleep

Stage 1 Sleep. EEG highlighted by red box.
Stage 2 Sleep. EEG highlighted by red box. Sleep spindles highlighted by red line.Studies of human sleep have established five well-defined stages, according to EEG recordings and polysomnography:

Non-REM sleep accounts for 75–80% of total sleep time:
Stage 1, with near-disappearance of the alpha waves seen in awake states, and appearance for the first time of theta waves. The stage is sometimes referred to as somnolence, or "drowsy sleep". It appears at sleep onset (as it is mostly a transition state into Stage 2) and is associated with the sudden twitches or hypnic jerks many people experience when falling asleep. While these are normal and of no concern, the hypnagogic hallucinations which some people may experience at this stage can be more troublesome. During this period, the subject loses some muscle tone, and conscious awareness of the external environment: Stage 1 can be thought of as a gateway state between wake and sleep.
Stage 2, with "sleep spindles" (12–16 Hz) and "K-complexes." The EMG lowers, and conscious awareness of the external environment disappears. This occupies 45–55% of total sleep.
Stage 3, with delta waves, also called delta rhythms (.5–4 Hz), is considered part of slow-wave sleep (SWS) and functions primarily as a transition into stage four. Overall it occupies 3–8% of total sleep time.
Stage 4 is true delta sleep. It predominates the first third of the night and accounts for 10–15% of total sleep time. This is often described as the deepest stage of sleep; it is exceedingly difficult to wake a subject in this state. This is the stage in which night terrors, bed wetting, and sleepwalking occur.

Stage 4 Sleep. EEG highlighted by red box.
REM Sleep. EEG highlighted by red box. Eye movements highlighted by red line.REM sleep is popularly associated with dreaming, especially bizarre, visual, and seemingly random dreams; however, dreams can also occur during sleep onset (hypnogogia) and during all stages of Non-REM sleep. REM sleep is predominant in the final third of a sleep period; its timing is linked to circadian rhythm and body temperature. The EEG in this period is aroused and looks similar to stage 1, and sometimes includes beta waves. Also known as Stage 5 sleep.
Sleep proceeds in cycles of NREM and REM phases. In humans, the cycle of REM and NREM is approximately 90 minutes. Each stage may have a distinct physiological function. Drugs such as alcohol and sleeping pills can suppress certain stages of sleep (see Sleep deprivation). This can result in a sleep that exhibits loss of consciousness but does not fulfill its physiological functions.

Each sleep stage is not necessarily uniform. Within a given stage, a cyclical alternating pattern may be observed.

Restorative theories of sleep describe sleep as a dynamic time of healing and growth for organisms. For example, during stages 3 and 4, or slow-wave sleep, growth hormone levels increase, and changes in immune function occur. Sleep deprivation, in some studies, leads to decrements in immune function and can alter metabolism under extreme, extended sleep deprivation paradigms. However, short periods of sleep deprivation have not been conclusively shown to significantly impact organ, muscular, cardiac, or other somatic function in ways that suggest that any of these systems are primarily influenced by sleep. Overall there is currently only scant evidence in support of the restorative hypothesis.
Non-REM sleep may be an anabolic state marked by physiological processes of growth and rejuvenation of the organism's immune, nervous, muscular, and skeletal systems (but see above). Sleep might restore neurons and increase production of brain proteins and certain hormones. Wakefulness may perhaps be viewed as a cyclical, temporary, hyperactive catabolic state during which the organism acquires nourishment and procreates. Also, during sleep, an organism is vulnerable; when awake it may perceive and avoid threats. Asking the question "Why do we awaken?" instead of "Why do we sleep?" yields a different perspective toward understanding how sleep and its stages contribute to a healthy organism.
According to the ontogenetic hypothesis of REM sleep, the activity occurring during neonatal REM sleep (or active sleep) seems to be particularly important to the developing organism (Marks et al., 1995). Studies investigating the effects of deprivation of active sleep have shown that deprivation early in life can result in behavioral problems, permanent sleep disruption, decreased brain mass (Mirmiran et al. 1983), and an abnormal amount of neuronal cell death (Morrissey, Duntley & Anch, 2004). Given sleep's heterogeneous nature, however, no single theory predominates, as it is difficult to describe one single "function" of sleep.
One process commonly believed to be highly dependent on sleep is memory. REM sleep appears to help with the consolidation of spatial and procedural memory, while slow-wave sleep helps with the consolidation of declarative memories. When experimental subjects are given academic material to learn, especially if it involves organized, systematic thought, their retention is markedly increased after a night's sleep. Mere rote memorization is retained similarly well with or without an intervening period of sleep. Some memory theorists argue that saving memory directly into long-term memory is a slow and error prone process, and propose that cerebral input is saved first in a temporary memory store, and then encoded and transferred into long-term memory during sleep. (Zhang, 2004).

Despite an abundance of positive findings in support of these ideas, many sleep scientists do not believe that sleep's primary function is related to memory. These scientists point out that many of the studies cited by proponents of this theory are contradictory or confounded by the side-effects caused by the experimental manipulations. A more salient issue is that only a handful of studies have shown that sleep actually influences brain plasticity--which is the mechanism underlying remembering and forgetting (Benington and Frank, 2003).
One view, "Preservation and Protection", is that sleep serves an adaptive function. It protects the individual during that portion of the 24-hour day in which being awake, and hence roaming around, would place the individual at greatest risk. Organisms don't require 24 hours to feed themselves and meet other necessities. From this perspective of adaptation, organisms are safer by staying out of harm's way where potentially they could be prey to other stronger organisms. They sleep at times that maximize their safety, given their physical capacities and their habitats. (Allison & Cicchetti, 1976; Webb, 1982). This theory, however, is not universally accepted. For example, if true, there would be no reason for the brain to disengage from the external environment as it does during normal sleep. A more advantageous adaptation would be for animals to seclude themselves but maintain quiet wakefulness to avoid predation. The fact is that sleep is itself a "drive" in that animals will alter their behaviors in order to obtain sleep: sleep is not simply a passive consequence of removing the animal from the environment. Therefore, circadian regulation is more than sufficient to explain periods of activity and quiescence that are adaptive to an organism but the more peculiar specializations of sleep most likely reflect different and unknown functions.
These several theories are not mutually exclusive; each may contain truths that will be validated in the future. Despite decades of intense research, scientists still have only clues to sleep function