Why do scientist think mad cow is from the feed when its really no different than manganese madness?

Question

I think the government scientists have blinders on to miss this connection.

http://www.bcm.edu/neurol/challeng/pat50/summary.html

ex-steel foundry worker

Do a google search on manganese madness and the gait disturbances and the halcinations and strange behavior. I beleive it having worked with very high levels of manganese.

what do you think is in that fertilizer they are puting on their fields that the cows are grazing on? Fertilizer contains manganese.

http://www.thestar.co.za/index.php?fSectionId=129&fArticleId=2396886

As for prions, here's another explanation under dissent.

http://en.wikipedia.org/wiki/Prion

Answer 1

Mad cow disease is more correctly called bovine spongiform encephalopathy. The name indicates its primary effect (rather than its symptoms), which is that the brain actually becomes "spongy" as the disease progresses.

Nothing in your link indicates a tendency of the brain to become spongy -- the deterioration of the basal ganglia referred to there is quite different from the unique sponge-like deterioration that occurs in BSE victims.

Moreover, the cause of BSE has been isolated -- it is a prion, a specific type of protein that's an even more primitive life form (if in fact it even IS a life form) than viruses. Prions are stored in the central nervous tissue and transferred from one host to another when the central nervous tissue is eaten. The mechanism is well known in cattle, sheep (where it is called scrapie), squirrels, and also in humans (where it is called Creutzfeldt Jakob Disease, or CJD; it is also known in Papua New Guinea as kuru).

Answer 2

I think they have identified the linkage between cattle showing symptoms of the mad cow disease having the BSE (bovine spongiform encephalitis) virus or bacteria in their system. They have also determined that if cattle or even humans who were previously healthy are infected with BSE, they come down with the symptoms. The reason the feed has been identified as a problem is because in the past, mad cow cattle have been re-processed into feed, which results in a new set of cattle getting the disease. So the agent causing mad cow has been clearly shown to be communicable, but "manganese madness" probably would not be.

Answer 3

It is different, because it it caused by self-replicating proteins called "prions". Manganese is a mineral. I am not sure from the description I read that the symptoms are even all that similar.

Answer 4

They KNOW it's from the feed. They KNOW that eating contaminated meat can transfer it to people.... Getting the British Govt in the late eighties to finally admit the overwhelming evidence to support this was a farce, to say the least.

BSE looks like Scrapie because that's basically what it is!

Wonder where J Selwyn-Gummer is now? He was the guy that forced his 2 daughters to eat hamburgers for the cameras while he was Minister for Agriculture! Hope the pompous twit was handsomely rewarded for his efforts!

Answer 5

I guess the assays that show the presence of prions is the major difference. The symptoms are the same but the cause is not the same.

Aloha

Answer 6

I blame it on the Huckleberry Railroad system, the grass along the rocks is so brittle and brown. I Love Mad Cows!!

Answer 7

Because cows don't do much alloy-welding. They do however eat feed containing eye and neural tissues exposing them to prion proteins.

Answer 8

just dont eat cow f*ck
or spinach

Answer 9

i think you are soo right.